Workshop-Synthetic Peptides for Biological Studies: Minimizing Peptide Degradation.


Chaired by Gregg Fields.

Presentations by


The development of "peptide drugs" has often been hindered by the relatively short half-life of peptides in vivo. In particular, peptides are hydrolyzed readily by enzymes such as amino- and carboxy-peptidases. Peptide degradation may be minimized by one of two simple, general approaches: conformational constraint of the peptide sequence and incorporation of D-amino acid analogs. Conformational constraints limit the peptide from assuming a vast array of flexible states, which potentially reduce the probability of the peptide "fitting" enzyme binding pockets. Constraints may be introduced by disulfide bridging or by lactamization using amino acid sidechains or free peptide amino and/or carboxy termini. Orthogonal approaches, compatible with the mild conditions of Fmoc chemistry, have made the introduction of these constraints possible with resin-bound peptides. The solid-phase mode may be advantageous due to a pseudo-dilution phenomenon, a kinetic effect that favors intramolecular reactions over intermolecular reactions (cyclodimerizations and cyclo-oligomerizations). Methods for on-resin (heterodetic) disulfide bond and (homodetic) lactam formation will be described. This discussion will focus on the requirements for an extra level of selectively cleavable protecting groups and anchoring linkages. The introduction of D-amino acid analogs to inhibit enzymatic degradation is straightforward but creates a potential problem of altering the desired recognition of the peptide. Two strategies for using D-amino acids, assembling D-enantiomer "mirror image" peptides and reverse-sequence all-D "retro-inverso" peptides, will be discussed. In particular, examples of both the success and failure of using D-amino acid peptides will be given, as well as potential guidelines for when each strategy may be applicable.

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Created: 27th July 1995
Last modified: 27th July 1995